DIABETES MELLITUS JUVENIL EM CÃO SEM RAÇA DEFINIDA – RELATO DE CASO

Abstract

Juvenile Diabetes mellitus is very rare and represents less than 1% of all cases of canine diabetes diagnosed, mainly due to hypoplasia and aplasia of the pancreatic islets, in addition to idiopathic atrophy of the pancreas. In dogs, type 1 DM predominates, related to the incapacity of insulin production by the endocrine pancreas, and may also occur in concomitance with other conditions that exacerbate clinical signs or make treatment difficult, such as inflammatory and endocrine diseases. The basic treatment consists of insulin therapy, dietary control, reduction of body weight, if the dog is overweight, daily exercise, cessation of diabetogenic drugs and control of simultaneous or underlying problems. This report describes a case of juvenile Diabetes mellitus in a male dog, eight months old, mixed breed, taken to the Veterinary Hospital of the CCA of the UFPB. The animal had the four classical clinical signs of DM, polyuria, polydipsia, polyphagia and weight loss. It also had abdominal tenderness, alopecia areas in the neck, back and limbs, hyper pigmentation, pustules, epidermal collars, comedones in the abdominal region and bilateral immature cataract. In the complementary exams, hyperglycemia, elevated glycosuria, mild ketonuria, hypostenuria, elevated ALT concentrations, serum albumin and total protein, mild hypochromic normocytic arregenerative anemia, mild relative and absolute neutrophilia, and relative lymphopenia were observed. In addition, he still had Demodex canis infection. Treatment with NPH insulin, 0.35 IU / kg, subcutaneously, BID, was instituted immediately after the feeding that was modified to a diet high in fiber and starch, in addition to daily walks of 20 to 30 minutes duration and treatment of concomitant conditions. The animal had a significant improvement, but there was still no stabilization of the glycemic curve, and it is necessary, also, correction in the therapy and investigation of possible factors that alter the insulin response.